Physical mechanisms shaping the Drosophila dorsoventral compartment boundary, Curr Biol, vol.22, pp.967-976, 2012. ,
ER stress activates the TOR pathway through Atf6, J Mol Signal, vol.13, 2018. ,
The Drosophila TNF receptor Grindelwald couples loss of cell polarity and neoplastic growth, Nature, vol.522, pp.482-486, 2015. ,
URL : https://hal.archives-ouvertes.fr/hal-01255294
Chemical stresses fail to mimic the unfolded protein response resulting from luminal load with unfolded polypeptides, J Biol Chem, vol.293, pp.5600-5612, 2018. ,
Endoplasmic reticulum and the unfolded protein response: dynamics and metabolic integration, Int Rev Cell Mol Biol, vol.301, pp.215-290, 2013. ,
A versatile PhiC31 based reporter system for measuring AP-1 and Nrf2 signaling in Drosophila and in tissue culture, PLoS One, vol.7, 2012. ,
Mechanism of Ca2+ disruption in Alzheimer's disease by presenilin regulation of InsP3 receptor channel gating, Neuron, vol.58, pp.871-883, 2008. ,
Defective intracellular transport is the molecular basis of rhodopsin-dependent dominant retinal degeneration, Proc Natl Acad Sci U S A, vol.92, pp.3070-3074, 1995. ,
Secreted peptide Dilp8 coordinates Drosophila tissue growth with developmental timing, Science, vol.336, pp.582-585, 2012. ,
URL : https://hal.archives-ouvertes.fr/hal-00766336
The PERK pathway independently triggers apoptosis and a Rac1/Slpr/ JNK/Dilp8 signaling favoring tissue homeostasis in a chronic ER stress Drosophila model, Cell Death Dis, vol.5, 2014. ,
Simons K (1995) CDC42 and Rac1 control different actin-dependent processes in the Drosophila wing disc epithelium, J Cell Biol, vol.131, pp.151-164 ,
Imaginal discs secrete insulin-like peptide 8 to mediate plasticity of growth and maturation, Science, vol.336, pp.579-582, 2012. ,
Perk is essential for translational regulation and cell survival during the unfolded protein response, Molecular Cell, vol.5, issue.5, pp.897-904, 2000. ,
The Nterminus of presenilin-2 increases single channel activity of brain ryanodine receptors through direct protein-protein interaction, Cell Calcium, vol.44, pp.507-518, 2008. ,
CDK5 and MEKK1 mediate proapoptotic signalling following endoplasmic reticulum stress in an autosomal dominant retinitis pigmentosa model, Nat Cell Biol, vol.14, pp.409-415, 2012. ,
) A drosophila reporter for the translational activation of ATF4 marks stressed cells during development, PLOS ONE, vol.10, issue.5, p.126795, 2015. ,
The pathogenic human Torsin A in Drosophila activates the unfolded protein response and increases susceptibility to oxidative stress, BMC Genomics, vol.16, p.338, 2015. ,
The selector gene apterous and notch are required to locally increase mechanical cell bond tension at the Drosophila dorsoventral compartment boundary, PLoS One, vol.11, 2016. ,
Intracellular calcium deficits in Drosophila cholinergic neurons expressing wild type or FAD-mutant presenilin, PLoS One, vol.4, 2009. ,
The cytosolic N-terminus of presenilin-1 potentiates mouse ryanodine receptor single channel activity, Int J Biochem Cell Biol, vol.40, pp.84-97, 2008. ,
Drosophila as a model for unfolded protein response research, BMB Rep, vol.48, pp.445-453, 2015. ,
Unfolded protein response in a Drosophila model for retinal degeneration, EMBO J, vol.26, pp.242-252, 2007. ,
The xbp-1 gene is essential for development in Drosophila, Dev Genes Evol, vol.217, pp.159-167, 2007. ,
URL : https://hal.archives-ouvertes.fr/hal-00125444
Enhanced ryanodinemediated calcium release in mutant PS1-expressing Alzheimer's mouse models, Ann N Y Acad Sci, vol.1097, pp.265-277, 2007. ,
Reinitiation involving upstream ORFs regulates ATF4 mRNA translation in mammalian cells, Proc Natl Acad Sci, vol.101, issue.31, pp.11269-11274, 2004. ,
The impact of the unfolded protein response on human disease, J Cell Biol, vol.197, pp.857-867, 2012. ,
The defender against apoptotic cell death 1 gene is required for tissue growth and efficient N-glycosylation in Drosophila melanogaster, Dev Biol, vol.420, pp.186-195, 2016. ,
, Publisher's note Springer Nature remains neutral with regard to jurisdictional claims in published maps and institutional affiliations